July 2015


Options: Use Proportional Font
Show HTML Part by Default
Show All Mail Headers

Message: [<< First] [< Prev] [Next >] [Last >>]
Topic: [<< First] [< Prev] [Next >] [Last >>]
Author: [<< First] [< Prev] [Next >] [Last >>]

Print Reply
Mitchel Cohen <[log in to unmask]>
Reply To:
Science for the People Discussion List <[log in to unmask]>
Sun, 5 Jul 2015 12:37:51 -0400
text/plain (49 kB) , text/html (51 kB)

Herd Immunity: Flawed Science and Mass Vaccination Failures

by <>Suzanne Humphries, MD

The oft-parroted sound bite – "we need herd 
immunity"- implies that if ninety five percent of 
the population can become "immune" to a disease 
target immunity levels will be met and diseases 
will either be eradicated or controlled. This 
sound bite is the most commonly pulled weapon 
used by the vaccinators, only second to "smallpox 
and polio were eradicated by vaccination." "Herd 
immunity" is the trump card for the defense of 
vaccination on TV, Internet, medical journals and 
newspapers as to why we should be vaccinated over 
and over throughout our lives, with an ever-increasing number of vaccines.

Paul Offit smiled 
PLAYED THE CARD while peddling his book on the 
comedy central channel as Steven Colbert jokingly 
said, "if the vaccines work so good for you, why 
do I need one?" 
Mark Segal 
IT on fox news as Mary Holland, JD eloquently 
described the issue of vaccine injury and loss of 
legal recourse in an era of forced and mandated 
vaccines. In addition to flaunting several false 
allegations and sound bites, Dr. Segal's 
well-rehearsed rant brushed right over the issue 
at hand, the fact that victims of vaccine injury 
have no legal right to sue – and instead launched 
into his agenda of scaring the listeners by 
parroting the "herd immunity" dogma.

The hype about herd immunity unfortunately 
creates a wall of hostility between those who 
vaccinate and those who delay some vaccines, 
avoid certain vaccines, or quit vaccinating altogether.

Since the beginning of vaccination, there is 
little proof that vaccines are responsible for 
eradicating disease even when herd immunity 
vaccination levels have been reached. Yet 
celebrity doctors rattle on about your 
unvaccinated neighbor being the biggest threat to 
your child – as if vaccination was the only way 
to avoid an illness or stay healthy.

To make matters worse, this intimidation to 
vaccinate is played out in an environment where 
WHO and vaccine manufacturers have been accused 
of scandalous misrepresentations of disease risk 
or vaccine safety and effectiveness. If the 
allegations against these entities are true, 
which I believe they are, we are being 
systematically altered, sickened and manipulated 
by powerful governing bodies that either don't 
understand the risks of vaccination, or don't 
care. We are told that the health of the herd is 
more important than any single life, and you now 
have no conventional legal recourse when your 
little sheep is wounded by any type of vaccine, no matter how it happened.

The Money Factor

The population of the world is expanding over the 
past 200 years where vaccines have been used, and 
this makes obtaining herd immunity even more 
expensive and impossible today than ever. How 
many billions of people would need to be 
vaccinated how many times to eradicate just one 
illness based on the theory of vaccine herd 
immunity? How much would that cost? Consider the 
cost of vaccines, refrigeration, vaccinators, and 
hazardous waste removal. Just look at chicken pox 
vaccine at $7.25 per dose for the CDC discounted 
price. Each child gets 2 doses.

The US census shows 25.7 million children between 
0-5 years. Just the cost of the vaccines to 
vaccinate each of those children, not including 
the lifetime of boosters, refrigeration, 
administration and waste, costs the government 
over 372 million dollars. Chicken pox vaccines 
are now being exposed for the failure they are, 
but vaccine profits are still climbing. After the 
members of the herd stopped transmitting natural 
immunity to each other because of the vaccine 
effect, shingles increased. The response -- more 
doses of vaccine for children and a shingles 
vaccine to adults. There is a recent journal 
abstract describing the failure of herd 
protection by 
vaccines. In a 
DOCUMENT, Dr. Goldman says:

"Prior to the universal varicella vaccination 
program, 95% of adults experienced natural 
chickenpox (usually as school aged children) -­ 
these cases were usually benign and resulted in 
long term immunity. This high percentage of 
individuals having long term immunity has been 
compromised by mass vaccination of children which 
provides at best 70 to 90% immunity that is 
temporary and of unknown duration -- shifting 
chickenpox to a more vulnerable adult population 
where chickenpox carries 20 times more risk of 
death and 15 times more risk of hospitalization 
compared to children. Add to this the adverse 
effects of both the chickenpox and shingles 
vaccines as well as the potential for increased 
risk of shingles for an estimated 30 to 50 years 
among adults. The Universal Varicella 
(Chickenpox) Vaccination Program now requires 
booster vaccines; however, these are less 
effective than the natural immunity that existed 
in communities prior to licensure of the varicella vaccine."

In India, doctors are concerned about profit 
margins being protected before human lives, with 
recommendations to vaccinate every child with 
more expensive, newer vaccines. 
Jacob Puliyel describes the problems he 

"An analysis in the Lancet showed how the 
Pneumococcal vaccine reduces only 4 cases of 
pneumonia per 1000 children. The cost for 
vaccinating 1000 children comes to $ 12,750. 
Treating the 4 cases of pneumonia in India using 
WHO protocol, would cost $ 1. The pneumococcus 
strains prevalent in India are nearly all 
sensitive to inexpensive antibiotics like 
penicillin. In the US which has been using the 
pneumococcal vaccine for some years now, there 
has been a strain shift – strains covered in the 
vaccine are being replaced by other strains. 
Ominously the new strains are more antibiotic 
resistant. Vaccine has simply made the problem of 
pneumococcal disease worse. Yet this vaccine is 
being pushed in Africa and Asia....It is not 
about lives lost in poor countries – it is all 
about the cash register. These organizations and 
their sponsors have profit margins to protect. 
Ethics is not a major issue with them."

The profits to vaccine manufacturers and the government must be enormous.

The CDC is in the vaccine business. Members of 
the CDC's Vaccine Advisory Committee accept 
payment from vaccine manufacturers. 
Sanofi-Pasteur, Merck and others specifically 
seek to employ CDC staff once their contracts 
have run out. Relationships have included sharing 
a vaccine patent, owning stock in a vaccine 
company, payments for research, payment to 
monitor manufacturer vaccine tests, and funding 
academic departments. Thanks to a 1980 law, the 
CDC currently holds dozens of licensing 
agreements. It also has numerous ongoing projects 
to collaborate on new vaccines.

The science?

What science is there behind the belief that the 
herd can be protected by vaccinating enough of 
the sheep? Or that any disease has been 
eradicated from the planet thanks to a vaccine?

Recently, I was told by a vaccinator that "herd 
immunity is just a definition and so it can't 
actually be wrong. " But the assumption of a 95% 
vaccination rate giving the herd a chance at 
eradication or higher levels of health – can be 
wrong. Let us go back in time and see just where 
the idea behind this definition probably comes 
from. Dr A.W. Hedrich in 1929, studied the natural occurrence of measles.

"On the basis of field surveys of various 
workers, it is inferred that approximately 95% of 
the children in cities suffer measles attacks by the fifteenth birthday. " [1]

Before vaccines, outbreaks of measles were 
observed in 2 to 3 year cycles, and 95% of the 
population developed immunity by the age of fifteen.

The original idea that vaccination could 
strengthen the herd's immunity, assumed that 
there was only one clinical event, and that one 
natural exposure equated life-long immunity. But 
this was not the case back when the diseases 
circulated freely. Vaccinators miss the point 
that the body defends most efficiently as a 
of ongoing re-exposure.

They try to mimic this with boosters. But the 
vaccination plan leaves the elderly (due to 
vaccine-induced immunity being short-lived and 
antigens taken out of circulation) and the very 
young (due to lack of transferrable maternal 
immunity) more vulnerable to several diseases 
that were not a threat to them before 
vaccination. In the case of chicken pox, 
vaccination renders the elderly more apt to 
shingles infections, because the herd has now 
lost the continued and benign re-exposures to children with chicken pox.

Instead of figuring out why a very small number 
develop dangerous invasive conditions, vaccine 
enthusiasts recommend vaccinating as often as 
possible in order to protect against something 
that would never be a danger to the vast majority 
of those vaccinated. If you constantly swab 
throats of healthy people most would be carrying 
and circulating supposed pathogens, as 
commensals.[2] At any one time in any society, 
neisseriae (the bacteria isolated in some cases 
of meningitis) are being circulated, yet most of 
the time, nothing happens, other than the body 
notes it, defends against it, and the host has no 
idea that they even carried it.[3] But now that 
vaccines for as many types as possible have been 
developed, the vaccine is the answer to the 
problem. This is typical for diseases today.


It is well documented that prior to vaccination, 
cycles of natural infection added to the herd's immunity.

"The formal demonstration that both maternal 
antibodies and early exposure to infection are 
required for long-term protection illustrated 
that constant re-infection cycles have an 
essential role in building a stable herd immunity.

In a population that is not constantly exposed to 
the infection during early infancy under the 
immunologic umbrella of maternal antibodies or 
vaccinated thoroughly a serious risk of re-emerging infections may arise. " [4]

Vaccination creates a "quasi-sterile" environment 
that opens up the possibility of disease outbreaks.

"Attempts to eradicate measles virus or 
poliovirus eliminates antigen exposure of infants 
to these pathogens. Such quasi-sterile 
epidemiological situations may actually increase the risk of outbreaks." [5]

We know this is possible because there have been 
eruptions of measles in the USA in populations 
that were 100 percent vaccinated.

"The affected high school had 276 students and 
was in the same building as a junior high school 
with 135 students. A review of health records in 
the high school showed that all 411 students had 
documentation of measles vaccination on or after 
the first birthday, in accordance with Illinois law." [6]

Within the scope of vaccination, when a 
quasi-sterile situation is created, and measles 
breaks out in the midst, the only solution within 
that paradigm is to vaccinate more people, more 
often. This is a backwards solution to the 
problem when considering who remains susceptible 
even in the face of full compliance: infants and 
non-immune adults. Susceptible age groups have 
essentially traded places since vaccinating.

What used to happen with measles is that infants 
were protected by maternal antibodies, adults 
were protected by continued exposure, and 
infected children handled the disease normally 
and became immune for long periods of time. So, 
while measles vaccines have decreased the 
expression of measles infections, it has not 
necessarily improved the bigger picture. And 
certainly there are numerous troubles with the side effects of the vaccine.

Prior to vaccination, mothers were naturally 
immune to measles and passed that immunity to 
their infants via placenta and breast milk. 
Vaccinated mothers may have vaccine immunity, 
which is not the same immunologically, as natural 
immunity. One of the major differences in the 
vaccine-induced immunity is that it cannot be passed from mother to infant.

Since most vaccines are delivered by injection, 
the mucous membranes are bypassed and thus blood 
antibodies are produced but not mucosal 
antibodies. Mucosal exposure is what contributes 
to the production of antibodies in the mammary 
gland. A child's exposure to the virus while 
being breastfed by a naturally immune mother 
would lead to an asymptomatic infection that 
results in long-term immunity to that virus. 
Vaccinated mothers have lower levels of 
virus-specific antibodies in the serum and milk 
compared to naturally immune mothers and thus their infants are unprotected.

"Infants whose mothers were born after 1963 had a 
measles attack rate of 33%, compared to 12% for 
infants of older mothers." Infants whose mothers 
were born after 1963 are more susceptible to 
measles than are infants of older mothers. An 
increasing proportion of infants born in the 
United States may be susceptible to measles." [7]

For the disease of measles, we see that while the 
clinical case rate may have declined with 
vaccination, the most sensitive members of the 
herd are at an increased risk -- as a result of vaccination.

Dr Peter Aaby has produced volumes of research on 
measles in Africa. Initially there was a belief 
that measles infection was associated with immune 
suppression and higher long-term mortality, but 
that belief came from vaccine research, not natural measles research.

"The belief in persistent immune suppression was 
stimulated by increased mortality after high-titre measles vaccination." [8]

Once natural measles was monitored long-term the 
knowledge changed. According to Aaby,

"When measles infection is mild, clinical measles 
has no long-term excess mortality and may be 
associated with better overall survival than no 
clinical measles infection. Sub-clinical measles 
is common among immunised children and is not 
associated with excess mortality." [9]

Measles is mildest when the infected person is 
replete with vitamins C and A. The devastation 
and mortality you hear about with measles comes from starving populations.

Do you know that 30% of cases of measles in 
unvaccinated are missed because they are so 
mild?[10] Subclinical measles is an entity that 
most doctors today are unaware of. If they are 
missed in unvaccinated, and there are known 
outbreaks of measles in 100 percent vaccinated 
populations, are cases missed in vaccinated 
populations too? Is measles still alive and well 
but going unnoticed in vaccinated countries, 
until a well-publicized outbreak occurs, as 
vaccine necessity is being trumpeted? What doctor 
would know or is even looking for atypical measles?

Talk to your grandmother about measles. Ask her 
if she saw death and destruction from the 
disease. It was not a disease that needed 
eradication. The high death rates were in 
countries where children were undernourished and 
lacked vitamins necessary to process the virus. 
Alexander Langmuir, MD is known today as "the 
father of infectious disease epidemiology." In 
1949 he created the epidemiology section of what 
is now known as the CDC. He also headed the Polio 
Surveillance Unit that was started in 1955 after 
the polio vaccine misadventures. Dr Langmuir knew 
that measles was not a disease that needed eradication when he said:

"To those who ask me, 'Why do you wish to 
eradicate measles?,' I reply with the same answer 
that Hillary used when asked why he wished to 
climb Mt. Everest. He said, 'Because it is 
there.' To this may be added, ". . and it can be done." [11]

Langmuir also knew that by the time vaccination 
was developed, measles mortality in the USA had 
already declined to minimal levels when he described measles as a

"... self-limiting infection of short duration, 
moderate severity, and low fatality..." [12]

The vaccine was created because it could be done, 
not because we needed it. Measles is not 
eradicated. Outbreaks happen all over the world, 
and will continue. And now infants will be 
unprotected because of the absence of maternal 
antibodies in their vaccinated mother's milk. So 
much for protecting the most vulnerable in the herd.


"We were fortunate enough to address their own 
medical (and) health officials where we reminded 
them of the incidence of smallpox in formerly 
"immunized" Filipinos. We invited them to consult 
their own medical records and asked them to 
correct us if our own facts and figures 
disagreed. No such correction has been 
forthcoming, and we can only conclude that 
between 1918-1919 there were 112,549 cases of 
smallpox notified, with 60,855 deaths. Systematic 
(mass) vaccination started in 1905, and since its 
introduction case mortality increased alarmingly. 
Their own records comment that "The mortality is 
hardly explainable."­Dr. Archie Kalokerinos from Second Thoughts on Disease

Orthopox is a member of the family of Poxviridae. 
The ancestor of the poxviruses is not known but 
structural studies suggest it may have been an 
adenovirus or a species related to both the 
poxviruses and the adenoviruses. Orthopox viruses 
include cowpox (vaccinia), smallpox (variola), 
and monkeypox. Mutations do occur in these viruses, but at a very slow rate.

Between October 1970 and May 1971 a poxvirus was 
isolated from some symptomatic patients in West 
Africa. That virus is now known as "human 
monkeypox." Monkeypox got its name because 
monkeys were the first animals known to have 
harbored the monkeypox virus. Scientists now say 
that the primary reservoirs for monkeypox virus 
are not monkeys but probably squirrels. WHO 
officials in 1976 had no idea what the true 
reservoir of infection was.[13] Today, according to CDC, it remains uncertain.

Smallpox was declared eradicated worldwide by the 
World Health Assembly on May 8th, 1980. 
Vaccination was stopped in the USA in 1972. 
However, poxviruses that were indistinguishable 
from smallpox continued to cause human disease.

Monkeys in surrounding areas where monkeypox 
outbreaks occur usually test negative for 
monkeypox. But prairie dogs, exotic rodents, 
Gambian rats, dormice, rope squirrels and other 
animals have tested positive. Nobody really knows 
when or where monkeypox viruses originated, but 
they seem to be close relatives of cowpox and 
smallpox. All three viruses have rodent 
reservoirs, which is important when considering 
the history and current transmission of smallpox 
and monkeypox. Today, monkeypox outbreaks are 
blamed on rodents or exotic pet imports, not 
person-to-person transmission even though human 
transmission does occur. Historically, smallpox 
reservoirs were also rodents – during a time when 
rodents were eaten as food and when infestations 
were commonplace. Yet in the discussion of 
smallpox outbreaks this is rarely mentioned. What 
we hear is how the vaccine eradicated the disease.

ARTICLE states that monkeypox was first recorded 
in 1970 after the eradication of smallpox in the 
Democratic Republic of Congo. University of 
California, School of Public Health 
epidemiologist Dr Anne Rimoin states that 
monkeypox first arrived in humans after smallpox 
eradication, even though it has been on the earth for millennia.

"Monkeypox has probably occurred for millennia in 
central Africa, but it's only since the 
eradication of smallpox that it's been a disease 
that actually happens in humans," Rimoin says. "

There is absolutely zero certainty as to when 
monkeypox first colonized humans. It is more 
accurate to say that monkeypox was first detected 
in humans around the time that smallpox was being 
declared eradicated, not that it arrived in 
humans at that time. Differentiation tests were 
not carried out on most cases of pox in the past 200 years.

Laboratory diagnostic assays for monkeypox 
include virus isolation and electron microscopy, 
ELISA, immunofluorescent antibody assay, 
histopathologic analysis, and Polymerase Chain 
Reaction (PCR). Unfortunately, most of these 
methods are relatively nonspecific and are unable 
to differentiate monkeypox viral infection from 
infection with other poxviruses.[14] All but PCR 
are fraught with false positives, false negatives, and cross reactivity.

In the 1970s and 1980s, biochemical tests were 
unreliable in differentiating between monkeypox 
and smallpox. Animal challenge tests were 
historically used to determine the difference 
between monkeypox and smallpox. The technique 
involved inoculating rabbits and watching the 
characteristics of the pox. Initially the two 
kinds of pox appear similar in the rabbit, but 
after a few days, monkeypox distinguishes itself 
as it becomes hemorrhagic. 

The problem with such means for distinction is 
that there has always been a hemorrhagic form of smallpox.

"There are four types of variola major smallpox: 
ordinary; modified; flat; and hemorrhagic.... 
Hemorrhagic smallpox has a much shorter 
incubation period and is likely not to be 
initially recognized as smallpox when presenting 
to medical care. Smallpox vaccination also does 
not provide much protection, if any, against hemorrhagic smallpox." [15]

ELISA is not much of a gold standard test as it 
casts a very wide net, and is fraught with false 
positive and false negative results.[16] 

The genomes of these three orthopox viruses are 
extremely conserved and require a technology that 
can detect the minute differences. Polymerase 
Chain Reaction (PCR) is a newer test that came on 
the scene in the 1980s. This test is different in 
that it can potentially find pieces of DNA from a 
virus. The genetic sequence of a virus has to 
first be mapped prior to designing a PCR test. So 
before smallpox, cowpox, or monkeypox viruses 
were characterized genetically, PCR could not be 
applied to distinguish between them.

The first PCR test for monkeypox was used in 
1997, but highly sensitive real-time PCR was not 
in use until 2006.[17] Different biotech 
companies have developed different tests that use 
different primers. PCR, while highly sensitive 
and specific at about 98%, still has drawbacks, 
contamination being the biggest one. No test is 
foolproof. Nonetheless it is probably the best 
assay available for detection and distinction today.

It should now be obvious that during the two 
centuries of smallpox vaccination and up until 
the 1990s there was no certain way of testing for 
distinct orthopox viruses. During the two 
centuries of vaccination, the viruses were likely 
to mutate, and certain strains could have been 
selected out as a result of vaccination.

Therefore, does anyone know how much 'smallpox' 
disease was actually monkeypox or vaccinia? Given 
that monkeypox is thought to be an ancient virus, 
where was it during the smallpox epidemics? Was it called hemorrhagic smallpox?

In 1972, scientists were asking similar questions when they said:

"Is it possible that there is an animal reservoir 
for smallpox infection? Could monkeypox be a 
source of new outbreaks of true variola? Or, can 
the monkeypox virus undergo certain mutations and 
become identical in its pathogenicity and 
infectiveness to the variola virus?" [18]

TO SCIENTIFIC AMERICAN, monkeypox is not that 
rare. Seven hundred and sixty cases of monkeypox 
were counted in the Congo between 2006 and 2007.

Before and during the time of eradication 
declaration, PCR was unavailable, and the 
different poxviruses couldn't be distinguished by 
their DNA, but by a skin test on rabbits, chick 
embryo membranes, and blood tests that were 
fraught with uncertainty. It seems to me that 
what was once called smallpox was likely a very 
non-uniform disease that could have been anything 
from cowpox to two forms of smallpox to chickenpox to monkeypox.

"Monkeypox virus is closely related to some other 
orthopoxviruses such as variola (smallpox) virus, 
and it cannot be distinguished from these viruses 
in some laboratory tests....In 1996-1997, an 
outbreak [of monkeypox] in the DRC continued for 
more than a year, with a person–to–person 
transmission rate estimated at 78%. However, 
epidemiological evidence suggests that many of 
the cases in this outbreak may have been 
chickenpox (varicella); the number of monkeypox 
cases and the transmission rate might have been 
overestimated due to self-reporting and the 
unavailability of laboratory testing." [19]

When vaccination stopped, monkeypox was suddenly 
diagnosed in humans. Diagnostic methods were 
absent during the great vaccine campaigns and 
everything pox-like was considered smallpox and 
counted as smallpox. Differentiating was not a priority.

Variola, the smallpox virus, is not in the 
smallpox vaccine. Instead, a cultured form of 
cowpox, called vaccinia, is the virus used to 
prevent smallpox. That same vaccine also covers 
monkeypox, according to the CDC:

Smallpox vaccine is effective at protecting 
people against monkeypox when it is given before 
they are exposed to monkeypox. (Exposure includes 
very close contact with a person or animal that 
has monkeypox.) Experts believe that vaccination 
after exposure to monkeypox may help prevent the 
disease or make it less severe." [20]

"Because the monkeypox virus is related to the 
virus that causes smallpox, the smallpox vaccine 
can protect people from getting monkeypox as well as smallpox.

Even though PCR can distinguish between the three 
viruses, clinically and immunologically the 
viruses are so similar, that one virus in the 
vaccine is thought to immunize against the two 
other viruses. During outbreaks they all look the same.

After the world trade center collapses in New 
York there were concerns over potential 
bioterrorism. Forty thousand health care workers 
and first responders and 450 thousand military 
were vaccinated in 2003. They were all contagious 
for the nineteen-day post-vaccine shedding 
period. Some doctors were asked to receive the 
vaccine in order to care for those who took the 
vaccine and developed vaccinia, or to care for 
those who became infected upon contact with a recently vaccinated person.

Multi-state outbreaks of monkeypox were reported 
in the same year.[21] Most cases are presumed to 
have come from contact with prairie dogs exposed 
to rodents per CDC. However all cases were not 
exposed to animals. 
TO A 2005 
of 72 cases only 37 cases were laboratory 
confirmed. Eleven original cases were thrown out 
of the database when they met exclusion criteria. 
CRITERIA. There is mention of human to human 
infection, though in some reports this is denied.

This is a very strange coincidence; vaccination 
and concomitant pox outbreaks in the same year. 
Supposedly, monkeypox is not easily transmissible 
between humans, but there is a report in the 
literature of a 5 chain human-to -human 
transmission, and human-to-human monkeypox 
transmission is well documented.[22] 
stated that "There was 'limited or no' spread of 
monkeypox virus through human contact during this outbreak."

In 2003, the year that half a million people were 
vaccinated in the USA – AND the only year of 
monkeypox outbreaks in the USA, a multistate 
(Illinois, Indiana, Kansas, Missouri, Ohio, and 
Wisconsin) outbreak, was the source of the 
outbreak definitely prairie dogs? CDC doesn't 
state how many pox cases were exposed to prairie 
dogs, just "the majority of them had direct or 
close contact." The vagueness of CDC's reports 
gives rise to doubts. Only 37 of 72 cases were 
confirmed with PCR tests, and eleven of the 
original total were excluded from analysis. 
Excluding numerous cases on frivolous grounds is 
one way to dampen a negative outcome after a vaccine accident.

Considering the link with vaccination is not 
far-fetched especially given that CDC reports say 
that only roughly half of cases were PCR 
confirmed. Vaccination has long been a relatively 
common means of transmitting pox outbreaks. 
According to Arita and Gromyko's WHO bulletin in 
1982, vaccination was a major fly in the eradication ointment...

"During the last 24 months, for example, 
surveillance reports from Canada and the United 
Kingdom have included 6 and 9 cases, 
respectively, of vaccine complications. At least 
8 cases, however, were in persons who, while not 
vaccinated themselves, had been infected with 
vaccinia virus after being in contact with 
persons recently vaccinated. In some countries 
vaccination of recruits to the armed services has 
continued; these recruits will occasionally 
transmit vaccinia infection to unvaccinated 
persons, and inevitably some of the complications 
will be fatal. In the United Kingdom and Finland, 
smallpox vaccination of army recruits was discontinued in 1981." [23]

Without discontinuing vaccination, it would have 
been impossible to stop the flow of smallpox. 
Doesn't that lead you to wonder how much smallpox 
was the result of the vaccine rather than natural 
smallpox? We know that in places like Leicester 
UK, when vaccination ceased, so did smallpox. And 
there are numerous accounts of smallpox disease 
not only being much more severe and deadly among 
vaccinated populations, but also more prevalent.

Isn't it interesting that smallpox vaccine defies 
everything we know about specificity in immunity 
and that one vaccine covers all sorts of pox, 
except chicken pox? Can you imagine, nowadays, if 
a vaccine researcher suggested that an illness 
could be prevented by using a slightly related 
virus? Today's vaccines contain numerous strains 
and types of the same organism. Polio vaccine has 
3 types of poliovirus, influenza 2 strains of 
type A and one strain of type B. But smallpox 
vaccine today contains one of many possible 
strains of a related virus, not even the 
smallpox(variola) virus at all. In Jenner's time, 
it is anyone's guess which viruses ended up in 
the vaccines since the technique was so primitive 
and typing methods were not available. Still, 
these vaccinia vaccines are thought to have 
eradicated smallpox, and serve as the foundation for vaccine faith.

Scientists back in the 1800s and early to mid 
1900s had no way to differentiate smallpox, 
cowpox, monkeypox or most other pox diseases in 
humans. Nor was there any effort to 
differentiate, until the disease was declared 
eradicated – just like when polio was eradicated. 
Anything that looked like polio, but not caused 
by a polio virus, was called acute flaccid paralysis.

Monkeypox and smallpox look identical on physical 
examination. Have a look at these two photos:


You probably can't tell the difference between 
the two diseases, and neither can most doctors. 
Edward Jenner and the doctors of the 1800s and 
1900s were also unable to distinguish smallpox – 
major and minor, monkeypox, or cowpox, or even chickenpox.

"When [monkeypox]infection in human beings does 
occur, it can be clinically indistinguishable 
from smallpox, chickenpox, and other causes of a vesiculopustular rash."


It is now known that many cases of smallpox were 
mild. These are termed variola minor. Variola 
major and variola minor are indistinguishable 
using the sensitive PCR test,[24] and thus 
represent the same infectious organism.

Do you think your doctor would know a case of 
variola minor if he/she saw it? Or would it just 
be called chicken pox? Do you think your doctor 
would even think that it could be smallpox, given 
that smallpox is thought to be eradicated? There 
are clinical means to distinguish the difference, 
but few doctors think of it, and in the minor 
forms of smallpox it wouldn't matter anyway.

Many believe that smallpox was eradicated from 
the planet because of vaccination. I once 
believed this idea that was taught to me in 
medical school, and that all conventional doctors 
parrot as if they understood the history. With 
just a little research it becomes evident that 
even though smallpox seems to have disappeared, 
this was not the result of mass vaccination.

It is obvious that the vaccines of 1796-1900s 
were not purified or uniform, yet they serve as 
the foundation for successful vaccination. They 
were made on farms from scrapings of infected cow 
bellies, coarsely filtered, and mixed in 
glycerine. While today's vaccine product may be 
more meticulously manufactured, the CDC admits 
that the science behind even modern smallpox 
recommendations has been little more than a guess.

" on duration of protection and 
recommendations on periodicity of vaccinations 
are limited and based to a large extent on 
historic precedent and expert opinion used to 
develop previous ACIP recommendations for 
smallpox vaccination for laboratory workers using orthopoxviruses." [25]

And CDC has no idea what antibody titer is protective.

"The levels of antibody reported by these tests 
indicate only exposure, and the protective 
antibody titer against smallpox infection is unknown." [26]

They surmise that the vaccine provides high-level immunity for 3-5 years.

Here is a graph of smallpox vaccination deaths 
and smallpox disease deaths, from England spanning the years of 1906-1922.
Smallpox vs. Smallpox Vaccine Deaths

The vaccine-associated deaths are conspicuously 
high, at about half the rate of smallpox deaths.

Dr. Charles T. Pearce in his 1868 essay on vaccination wrote:

"It is a remarkable fact that Jenner's [the 
inventor of smallpox vaccine] first child, his 
eldest son, on whom he experimented, died 
subsequently of consumption[tuberculosis]. 
Another of his subjects, the man Phipps, whom 
Jenner vaccinated, also died of consumption."

Those who were vaccinated for smallpox were noted 
to be more severely affected by smallpox and 
tuberculosis. Many were exposed to tuberculosis 
from tuberculous animals that were used to make 

Smallpox manifested in several different 
forms(ordinary, modified, malignant, 
hemorrhagic). Genetically the minor and major 
forms of variola are related and 
indistinguishable by PCR. Individual 
susceptibility, rather than the virus probably 
made the biggest difference. Susceptibility would 
have certainly increased after injection of 
filthy vaccines that contained myriad bacteria and viruses.

What is most likely is that the appearance and 
disappearance of epidemics had much to do with 
the constitution and care of the population of 
the times. Scurvy was common in areas with 
hemorrhagic smallpox. This is no surprise to 
anyone who understands the full spectrum of 
ascorbic acid's function in the body, especially on blood vessels.

Pox epidemics declined as a result of sanitation 
and improved nutrition. During the era of 
smallpox most people were living in squalor, 
eating no fresh food, but rotten milk and rotten 
meat, drinking sewer water, living among filthy 
rodents, and working long hours for little pay. 
Pox viruses are ancient, but smallpox evolved as 
a deadly killer as humanity devolved to 
overcrowded city dwellers living with filth, squalor, and desperation.

Historical evidence points to the fact that the 
vaccinated were amongst the sickest in times of 
smallpox vaccines. Protests against the 
vaccinators and smallpox vaccination were 
massive.[27] Parents commonly chose jail rather 
than permit their newborn babies to be 
vaccinated. Entire towns and districts revolted 
before the disease was finally declared 
eradicated, and the vaccine madness ended.

Smallpox vaccination ended in the 1980s because 
smallpox had declined and because there was so 
much trouble with the old unsafe vaccine. That 
same trouble with the newer supposedly more safe 
smallpox vaccines is why smallpox vaccination 
ended after the 2003 first responder effort. 
Which makes you wonder just how much more trouble 
there was with the old smallpox vaccine which had 
a very long list of known bacterial and other 
"contaminants" because of its method of 
production. After the 2003 vaccines, reports of 
generalized vaccinia, autoinoculation, erythema 
multiforme, myopericarditis, ocular vaccinia, and 
postvaccinial encephalitis were reported.

Smallpox was declared eradicated before clear 
distinctions between different poxviruses were 
made using DNA analysis. Symptoms alone are what 
were counted for smallpox during smallpox 
epidemics. Vaccination was a major source of 
smallpox outbreaks, and only a small portion of 
the earth's entire herd was ever even vaccinated. 
Considering all of this, how can anyone believe 
that smallpox was eradicated with a vaccine?

With every vaccine suppressible disease, the 
general hysteria level usually depends on the 
availability of a vaccine. Once a vaccine was 
available, the disease was suddenly made out to 
be more problematic. Look how dangerous chicken 
pox became after the vaccine was developed.

Pertussis is now hot news and the unvaccinated 
interrupting herd immunity is raised over and 
over, despite the science that shows the 
vaccinated are by far and away the most affected by whooping cough.

"Our unvaccinated and under-vaccinated population 
did not appear to contribute significantly to the 
increased rate of clinical pertussis. 
Surprisingly, the highest incidence of disease 
was among previously vaccinated children in the 
eight to twelve year age group." [28]

This is the most recent, but not the first study 
to demonstrate 86% of cases of proven whooping 
cough are in the vaccinated. How can getting even 
100% vaccination uptake create an immune herd with such vaccines?

Mumps vaccine was known to be ineffective after 
two major outbreaks in vaccinated populations in 
the USA. Yet the solution was to double the 
boosters in children with a vaccine that is now 
by two former Merck scientists, to have been 
known to be ineffective by Merck's executives.

Jenner's initial promise was "We have a vaccine 
that will protect you for life with one 
injection." But even he was revaccinating his 
patients yearly, within 5 years of making that 
statement. And when that doesn't pan out with 
whooping cough, measles, mumps and whatever, the 
authorities say,, "We have a highly effective 
vaccine if it is given on time with boosters," 
then "This is an excellent vaccine when 3 or 4 
boosters are given, and adults are revaccinated." 
Or in the case of whooping cough, introducing an 
all-together new vaccine. There is a new nasal 
vaccine in the pipeline for newborns, which will 
be given alongside the already ineffective 
whooping cough vaccine series in childhood. This 
will no doubt be touted as a wonderful combination.

Eradication target dates are constantly moved 
forward, and the unvaccinated or the vaccine 
refusers are blamed for all outbreaks. Or in the 
case of Pakistan, they are branded 
FANATICS for not wanting their children to have 
30 oral polio vaccines by age 5. I have outlined 
in a 
BLOG, just what is really going on in India and 
how her people are being terrorized by WHO and 
CDC as the rate of paralysis continues to skyrocket.

I believe that when diseases disappear from 
sight, the disappearance is never solely by 
virtue of the vaccine. Yet the vaccine always 
gets the credit, because the blind faith in 
vaccines is prioritized over the scientific 
evidence. Evidence to the contrary of the value 
of vaccination is consistently snuffed out and 
kept away from the mainstream media, so that the 
herd never hears a peep of the truth. Instead, 
they get the "herd immunity" sound bite, which 
gives undeserved credit to the risk-benefit ratio 
of vaccination. Inside the web of half-truths and 
misinformation, the vaccine religion somehow 
justifies the public display of resentment and fear of the unvaccinated.

A special thank you to "O" from 
VACCINES" for assistance in editing this document.

1. Hedrich AW. 1930. The corrected average attack 
rate of measles among city children. Am. J. Epidemiol. 11 (3): 576-600.
2. Hjuler IM. 1995. Bacterial colonization of the 
larynx and trachea in healthy children. Acta 
Paediatr. 1995 May;84(5):566-8. PMID:7633155
3.Caugant DA. 2009. Meningococcal carriage and 
disease­population biology and evolution. 
Vaccine. 2009 Jun 24;27 Suppl 2:B64-70. PMID: 19464092
4. Navarini AA et al. 2010. Long-lasting immunity 
by early infection of maternal-antibody-protected 
infants. Eur J Immunol. Jan;40(1):113-6. PMID: 19877011
5. ibid. Navarini.
6. Measles Outbreak among Vaccinated High School 
Students – Illinois. MMWR. June 22, 1984 / 
7. Papania M. et al. 1999. Increased 
susceptibility to measles in infants in the 
United States. Pediatrics. Nov;1045(5):e59 pp 1-6. PMID 19545585.
8. Aaby P. et al. 2002. Low mortality after mild measles infection compared to
uninfected children in rural west Africa. 
Vaccine. Nov 22;21(1-2):120-6. PMID:12443670
9. ibid Aaby.
CHILDREN. Indian J Prev Soc Med. Vol 34 (1) pp 8-16.
11. Langmuir A.1962 .The importance of measles as 
a health problem. AJPH vol 52 no 2 pp1-4.
12. Ibid Langmuir.
13. Arita and Henderson. 1976. Monkeypox and 
whitepox viruses in West and Central Africa. Bull 
World Health Organ. 1976; 53(4): 347–353.
14. Weinstein Robert. 2005. Reemergence of 
Monkeypox: Prevalence, Diagnostics, and 
Countermeasures. Clin Infect Dis. 41 (12): 1765-1771.
15.US FDA. Vaccines, blood and biologics. 
16. Human anti-mouse antibodies (HAMA) are a 
common cause of false positive ELIZA. A person 
can develop HAMA for different reasons. The 
clinical use of monoclonal mouse antibodies 
(e.g., for radioimaging, in the treatment of some 
cancers) often produces HAMA. HAMA may also arise 
because of incidental or occupational exposure to 
foreign proteins (e.g. veterinarians, farm 
workers, food preparers) or due to the presence 
of domestic animals in the home environment. 
Blood transfusion and dialysis are among other 
sources of heterophilic antibodies.
18. Is monkeypox a reservoir of smallpox? 
December 25, 1972. JAMA. 1972;222(13):1645-1646.
19. Monkeypox. 2009. Center for food security and 
public health. Iowa state university. Pg 1-9.
20. CDC Fact Sheet. Smallpox vaccine and 
21. US CDC. MMWR. July 11, 2003 / 52(27);642-646. 
Update: Multistate Outbreak of Monkeypox ­ 
Illinois, Indiana, Kansas, Missouri, Ohio, and 
Wisconsin, 2003.
22. Fenner et al. 1989. Smallpox and its 
eradication. Page 1306.ISBN-10: 9241561106
23. Arita and Gromyko. Surveillance of 
orthopoxvirus infections, and associated 
research, in the period after smallpox. Bull 
World Health Organ. 1982; 60(3): 367–375. PMCID: PMC2536002 eradication
23A. Lancet Review. Jan 2004. Monkeypox. vol 4. pp 21-25.
24. Loveless BM. 2009. Differentiation of Variola 
major and Variola minor variants by MGB-Eclipse 
probe melt curves and genotyping analysis. Mol 
Cell Probes. 2009 Jun-Aug;23(3-4):166-70. Epub 
2009 Apr 5.
25. US CDC Emergency preparedness and response. 
CDC Interim Guidance for Revaccination of 
Eligible Persons who Participated in the US 
Civilian Smallpox Preparedness and Response 
26. US CDC. Emergency preparedness and response. 
Questions and Answers About Post-event 
27. Durbach, Nadja. 2004. Bodily Matters: The 
Anti-Vaccination Movement in England, 1853–1907. ISBN-10: 0822334127
28. Witt M et al. 2012. Unexpectedly Limited 
Durability of Immunity Following Acellular 
Pertussis Vaccination in Pre-Adolescents in a 
North American Outbreak. Clin Infect Dis. Clin 
Infect Dis. 2012 Jun;54(12):1730-5. PMID:22423127

Dr. Suzanne Humphries is a conventionally 
educated medical doctor who has taken the walk 
into, around, and out of the allopathic paradigm. 
She fully and successfully participated in the 
conventional system for 19 years, witnessing 
first-hand how that approach fails patients and 
creates new disease time and again. Prior to 
medical school, she earned a bachelor’s degree in 
physics from Rutgers University.

Dr. Humphries is on the board of directors of the 
Medical Council on Vaccination. She lives in Maine, USA.

Visit her website is <>

Mitchel Cohen's latest book, 
Bookchin! Commentaries on the Life and Thought of 
Murray Bookchin with conflicting views and essays 
by Mitchel Cohen, Brian Tokar, Chaia Heller, & 
John Clark, is now available for $8 measly bucks!

Also, click on the title to order a copy of 
Mitchel's book, 
Is Direct Action? (foreword by Richard Wolff) (596 pages). Get it now!

Mitchel's poetry books (click on the name): 
Cat Takes Flight and 
Permanent Carnival .

AND, please check out the latest at 
    * <>MONSANTO'S 
ROUNDUP Glyphosate found to be a likely Carcinogen
    * <>FREE 
LINA KHATTAB! 18-year-old student imprisoned and beaten, in Israel
    * <>DRONES 
& TARGETTED KILLINGS including interviews with Marjorie Cohn & Jeanne Mirer
Listen to 
by Mitchel Cohen: 
Permanent Carnival, and 
Memoriam: For Fallen 

    * <>IS 
Biological Determinism, by Mitchel Cohen

Ring the bells that still can ring,  Forget your perfect offering.
There is a crack, a crack in everything, That's how the light gets in.
~ Leonard Cohen

Realize that little things lead to bigger things 
... And there s a wonderful parable in the New 
Testament: The sower scatters seeds. Some seeds 
fall in the pathway and get stamped on, and they 
don t grow. Some fall on the rocks, and they don 
t grow. But some seeds fall on fallow ground, and 
they grow and multiply a thousandfold. Who knows 
where some good little thing that you ve done may 
bring results years later that you never dreamed of.
~ Pete Seeger