Agreed about cellulitis and even a foot infection causing that hyperemic type flow but the aggravating ones are the obvious
stenotic diabetic tibials, I wish they'd just cooperate and not compress at all...
On Tue, 1 Jun 2021 20:05:10 +0000, Audrey Fleming <[log in to unmask]> wrote:
>Can't cellulitis cause a low resistance monophasic waveform in a non-diseased artery? The increased perfusion creates a continuous flow waveform.
>
>Sent from Yahoo Mail on Android
>
> On Tue, Jun 1, 2021 at 8:03 PM, Harvey Wilson<[log in to unmask]> wrote: Very interesting folks! I was only hoping for a bit of speculation on tissue compressibility and got a bonus!
>We typically will fall back on getting waveforms if possible and a TBI - but as Bart states they are not always possible, those short, fat toes, etc. As for the toe clamp, we use tape still and if I need to get good PPG contact on those greasy ones, I will place Saran wrap very lightly around just to hold the PPG on without pressure.
>I have only tried a couple of times to get a PAT since watching a lecture on it at the NWVT. I find those little guys in the plantar foot quite tricky to find and our Radiologists and vascular surgeons might have never heard of it. Are PAT's well validated and widely used?
>Partially compressible, yeah, what to do with that possibility? What bugs me is to take obviously monophasic waveforms in the tibials but an ABI of 1.0 and then see it reported as "no evidence of arterial insufficiency" :( :(
>
>Harvey
>
>On Tue, 1 Jun 2021 11:10:22 -0700, Barton Bean <[log in to unmask]> wrote:
>
>>Hi Bill,
>>There are so many variables - cuff to toe sizes, what pressure is on the
>>PPG (very difficult to be precise in my experience) - except perhaps with
>>tape which we no longer use (clamp with unknown pressure is provided by
>>current equipment manufacturer), is the light on in the room, does the
>>patient have and small vessel calcifications.
>>Don't get me wrong they are a part of our protocol and we do them. I just
>>no longer believe the results particularly since a large part of our work
>>is with diabetic wound care.
>>PAT has been wonderful for us (even though it has limitations too).
>>Fond regards,
>>Bart
>>
>>On Tue, Jun 1, 2021 at 10:54 AM Bill Schroedter <
>>[log in to unmask]> wrote:
>>
>>> Hey Bart,
>>> Absolutely PAT's but could you please elaborate on your reservations re:
>>> toe pressures?
>>> Thanks,
>>> Bill
>>>
>>> -------- Original message --------
>>> From: Barton Bean <[log in to unmask]>
>>> Date: 6/1/21 1:41 PM (GMT-05:00)
>>> To: [log in to unmask]
>>> Subject: Re: Non compressiblility - tissue vs vessel
>>>
>>> PAT's (pedal acceleration times)are invaluable in a patient like this (and
>>> for that matter in our lab all wound care patients).
>>> In my experience ankle/toe pressure have some limited value but we are
>>> moving to PAT studies pre and post intervention.
>>> I cannot seem to be consistent with toe pressure even though I have
>>> performed thousands - I don't trust them any more.
>>> Best,
>>> Bart
>>>
>>> On Tue, Jun 1, 2021 at 4:35 AM Bill Schroedter <
>>> [log in to unmask]> wrote:
>>>
>>>> Good question Harvey. I agree with Denise. For wound care patients, many
>>>> of which are diabetic, this is an issue and toe pressures are really a
>>>> must.
>>>>
>>>> That said, I am trying to wrap my head around your question about the
>>>> effect of tissue edema – seems like that should transmit the pressure to
>>>> the vessel. Elaphantiasis Nostras would likely result in a similar
>>>> situation to that is much more common, lipodermatosclerosis. The resultant
>>>> tissue fibrosis / sclerosis in my mind would be more likely make a
>>>> difference in pressure transmission to the vessel. If you are 30-40 mmHg
>>>> above brachial pressure, no problem - your question is answered. But for
>>>> nearly my whole career I have wondered about what I call partially
>>>> compressible vessels – ones that are stiff and could in fact require more
>>>> than the transmitted tissue pressure to compress. What if the vessel in
>>>> fact does compress so that we obtain a falsely elevated pressure but is
>>>> impossible to determine the presence or degree – we really have no way of
>>>> knowing that. Our solution - when doing these, we always keep in mind that
>>>> we also have the Doppler waveform which should be strongly considered in
>>>> the interpretation of ankle pressures. A monophasic signal (or one with a
>>>> delayed upstroke) is unlikely to have a normal arterial pressure. The great
>>>> strength of our technology – a lot of clues – they should all point to the
>>>> same thing.
>>>>
>>>> Regards,
>>>>
>>>> Bill
>>>>
>>>>
>>>>
>>>> *From:* UVM Flownet <[log in to unmask]> *On Behalf Of *Denise Levy
>>>> *Sent:* Tuesday, June 1, 2021 2:09 AM
>>>> *To:* [log in to unmask]
>>>> *Subject:* Re: Non compressiblility - tissue vs vessel
>>>>
>>>>
>>>>
>>>> Hi,
>>>>
>>>> You will have more success with a metatarsal cuff and a digital toe cuff,
>>>> TBI using a PPG for the pressure.
>>>>
>>>> Very often ABI’s are falsely elevated due to diabetes plus 255 !
>>>>
>>>> The levels can be found on a search for TBI ranges.
>>>>
>>>> Thank you for your question,
>>>>
>>>> Denise Levy, RDMS, RVT
>>>>
>>>> Hill Vascular and Vein Center
>>>>
>>>> Vascular Lab Director
>>>>
>>>>
>>>>
>>>>
>>>>
>>>> On Mon, May 31, 2021 at 5:02 PM Bill Johnson <[log in to unmask]>
>>>> wrote:
>>>>
>>>> Harvey i do believe your question is quite valid!
>>>>
>>>> Personally I can only recount a few cases and I know there was a point
>>>> beyond which I would not increase the probe pressure. "Do no harm seemed a
>>>> good idea then and still.
>>>>
>>>> So, what do you do in these cases? I assume you do some type of
>>>> follow-up? Any lessons for us still learning?
>>>>
>>>>
>>>>
>>>> On Mon, May 31, 2021, 4:24 PM Harvey Wilson <[log in to unmask]>
>>>> wrote:
>>>>
>>>> Hi,
>>>>
>>>> I've often wondered about getting non comp ABI's on wound clinic patients
>>>> who have severe edema or something like Elaphantiasis Nostras. How much
>>>> does hardening and thickening of tissue contribute in cases like these vs
>>>> plain old calcification? I thought I'd post this here as I don't have
>>>> anyone here to bounce ideas off.
>>>>
>>>> Thanks,
>>>>
>>>> Harvey Wilson RVT RDMS RDCS
>>>> Victoria
>>>>
>>>> To unsubscribe or search other topics on UVM Flownet link to:
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>>>>
>>>> To unsubscribe or search other topics on UVM Flownet link to:
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>>>>
>>>> --
>>>>
>>>> Sent from Gmail Mobile
>>>>
>>>> To unsubscribe or search other topics on UVM Flownet link to:
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>>>
>>>
>>>
>>> --
>>> Barton A. Bean IV, RVT, FSVU
>>> Orange County Vascular Specialists
>>> 23236 Via Bahia
>>> Mission Viejo, CA 92691-2114
>>> E-mail [log in to unmask]
>>> (714)803-6288
>>> To unsubscribe or search other topics on UVM Flownet link to:
>>> http://list.uvm.edu/archives/uvmflownet.html
>>> To unsubscribe or search other topics on UVM Flownet link to:
>>> http://list.uvm.edu/archives/uvmflownet.html
>>
>>
>>
>>--
>>Barton A. Bean IV, RVT, FSVU
>>Orange County Vascular Specialists
>>23236 Via Bahia
>>Mission Viejo, CA 92691-2114
>>E-mail [log in to unmask]
>>(714)803-6288
>>
>>To unsubscribe or search other topics on UVM Flownet link to:
>>http://list.uvm.edu/archives/uvmflownet.html
>>
>
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