To unsubscribe or search other topics on UVM Flownet link to: http://list.uvm.edu/archives/uvmflownet.htmlHey Bart,Absolutely PAT's but could you please elaborate on your reservations re: toe pressures?Thanks,Bill-------- Original message --------From: Barton Bean <[log in to unmask]>Date: 6/1/21 1:41 PM (GMT-05:00)Subject: Re: Non compressiblility - tissue vs vesselPAT's (pedal acceleration times)are invaluable in a patient like this (and for that matter in our lab all wound care patients).In my experience ankle/toe pressure have some limited value but we are moving to PAT studies pre and post intervention.I cannot seem to be consistent with toe pressure even though I have performed thousands - I don't trust them any more.Best,BartTo unsubscribe or search other topics on UVM Flownet link to: http://list.uvm.edu/archives/uvmflownet.htmlGood question Harvey. I agree with Denise. For wound care patients, many of which are diabetic, this is an issue and toe pressures are really a must.
That said, I am trying to wrap my head around your question about the effect of tissue edema – seems like that should transmit the pressure to the vessel. Elaphantiasis Nostras would likely result in a similar situation to that is much more common, lipodermatosclerosis. The resultant tissue fibrosis / sclerosis in my mind would be more likely make a difference in pressure transmission to the vessel. If you are 30-40 mmHg above brachial pressure, no problem - your question is answered. But for nearly my whole career I have wondered about what I call partially compressible vessels – ones that are stiff and could in fact require more than the transmitted tissue pressure to compress. What if the vessel in fact does compress so that we obtain a falsely elevated pressure but is impossible to determine the presence or degree – we really have no way of knowing that. Our solution - when doing these, we always keep in mind that we also have the Doppler waveform which should be strongly considered in the interpretation of ankle pressures. A monophasic signal (or one with a delayed upstroke) is unlikely to have a normal arterial pressure. The great strength of our technology – a lot of clues – they should all point to the same thing.
Regards,
Bill
From: UVM Flownet <[log in to unmask]> On Behalf Of Denise Levy
Sent: Tuesday, June 1, 2021 2:09 AM
To: [log in to unmask]
Subject: Re: Non compressiblility - tissue vs vessel
Hi,
You will have more success with a metatarsal cuff and a digital toe cuff, TBI using a PPG for the pressure.
Very often ABI’s are falsely elevated due to diabetes plus 255 !
The levels can be found on a search for TBI ranges.
Thank you for your question,
Denise Levy, RDMS, RVT
Hill Vascular and Vein Center
Vascular Lab Director
On Mon, May 31, 2021 at 5:02 PM Bill Johnson <[log in to unmask]> wrote:
Harvey i do believe your question is quite valid!
Personally I can only recount a few cases and I know there was a point beyond which I would not increase the probe pressure. "Do no harm seemed a good idea then and still.
So, what do you do in these cases? I assume you do some type of follow-up? Any lessons for us still learning?
On Mon, May 31, 2021, 4:24 PM Harvey Wilson <[log in to unmask]> wrote:
Hi,
I've often wondered about getting non comp ABI's on wound clinic patients who have severe edema or something like Elaphantiasis Nostras. How much does hardening and thickening of tissue contribute in cases like these vs plain old calcification? I thought I'd post this here as I don't have anyone here to bounce ideas off.
Thanks,
Harvey Wilson RVT RDMS RDCS
Victoria
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--To unsubscribe or search other topics on UVM Flownet link to: http://list.uvm.edu/archives/uvmflownet.htmlBarton A. Bean IV, RVT, FSVUOrange County Vascular Specialists
23236 Via Bahia
Mission Viejo, CA 92691-2114
E-mail [log in to unmask]
(714)803-6288